Ataxic cerebral palsy

Ataxic cerebral palsy
The cerebellum (shown in red) is the region of the brain affected by ataxic cerebral palsy
SpecialtyNeurology

Ataxic cerebral palsy is clinically in approximately 5–10% of all cases of cerebral palsy, making it the least frequent form of cerebral palsy diagnosed.[1] Ataxic cerebral palsy is caused by damage to cerebellar structures, differentiating it from the other two forms of cerebral palsy, which are spastic cerebral palsy (damage to cortical motor areas and underlying white matter) and dyskinetic cerebral palsy (damage to basal ganglia).[2]

Because of the damage to the cerebellum, which is essential for coordinating muscle movements and balance, patients with ataxic cerebral palsy experience problems in coordination, specifically in their arms, legs, and trunk. Ataxic cerebral palsy is known to decrease muscle tone.[3]

The most common manifestation of ataxic cerebral palsy is intention (action) tremor, which is especially apparent when carrying out precise movements, such as tying shoe laces or writing with a pencil. This symptom gets progressively worse as the movement persists, causing the hand to shake. As the hand gets closer to accomplishing the intended task, the trembling intensifies which makes it even more difficult to complete.[4]

Like all forms of CP, there is no cure for ataxic cerebral palsy. However, there are a number of diverse treatments which together have been used to limit the negative effects of the condition. Like all forms of CP it is most common for ataxic cerebral palsy to be congenital, resulting from errors in the development of the cerebellum and connexins during pregnancy. However it is also possible to be acquired via meningitis or even by head trauma.[5]

  1. ^ Cite error: The named reference McHale was invoked but never defined (see the help page).
  2. ^ Cite error: The named reference Cheney was invoked but never defined (see the help page).
  3. ^ Straub, Kathryn.; Obrzut, John E. (2009). "Effects of cerebral palsy on neurophsyological function". Journal of Developmental and Physical Disabilities. 21 (2): 153–167. doi:10.1007/s10882-009-9130-3. S2CID 144152618.
  4. ^ Cite error: The named reference O'Shea was invoked but never defined (see the help page).
  5. ^ Cite error: The named reference Ingram was invoked but never defined (see the help page).