Pattern hair loss (also known as androgenetic alopecia (AGA)[1]) is a hair loss condition that primarily affects the top and front of the scalp.[2][3] In male-pattern hair loss (MPHL), the hair loss typically presents itself as either a receding front hairline, loss of hair on the crown and vertex of the scalp, or a combination of both. Female-pattern hair loss (FPHL) typically presents as a diffuse thinning of the hair across the entire scalp.[3]
Genetic research has identified alleles associated with male pattern hair loss.[4] These alleles appear to be undergoing positive sexual selection in European and East Asian populations, as male pattern baldness may be seen as a masculine attribute associated with seniority and higher social status.[4][citation needed] The condition is caused by a combination of male sex hormones (balding never occurs in castrated men) and genetic factors.[5]
The cause in female pattern hair loss remains unclear;[3] androgenetic alopecia for women is associated with an increased risk of polycystic ovary syndrome (PCOS).[13][14][15]
By the age of 50, pattern hair loss affects about half of males and a quarter of females.[3] It is the most common cause of hair loss. Both males aged 40–91[17] and younger male patients of early onset AGA (before the age of 35), had a higher likelihood of metabolic syndrome (MetS)[18][19][20][21] and insulin resistance.[22] With younger males, studies found metabolic syndrome to be at approximately a 4× increased frequency which is clinically deemed significant.[23][24] Abdominal obesity, hypertension and lowered high density lipoprotein were also significantly higher for younger groups.[25]
^ abcdefghVary JC (November 2015). "Selected Disorders of Skin Appendages--Acne, Alopecia, Hyperhidrosis". The Medical Clinics of North America (Review). 99 (6): 1195–1211. doi:10.1016/j.mcna.2015.07.003. PMID26476248.
^ abCite error: The named reference Hillmer Freudenberg Myles Herms 2009 pp. 255–264 was invoked but never defined (see the help page).
^Randall VA (2012-07-26). "Androgens and hair: a biological paradox with clinical consequences". Testosterone. Cambridge University Press. pp. 154–176. doi:10.1017/cbo9781139003353.008. ISBN978-1-139-00335-3. Male pattern baldness is androgen dependent, since it does not occur in castrates, unless they are given testosterone (Hamilton 1942), nor in XY individuals with androgen insensitivity due to non-functional androgen receptors (see Chapter 3). The genetic involvement in androgenetic alopecia is also pronounced.
^Cite error: The named reference pmid34668238 was invoked but never defined (see the help page).
^Huang J, Ran Y, Pradhan S, Yan W, Dai Y (2019). "Investigation on Microecology of Hair Root Fungi in Androgenetic Alopecia Patients". Mycopathologia. 184 (4): 505–515. doi:10.1007/s11046-019-00345-8. PMID31240449. S2CID195353938.
^Ertas R, Orscelik O, Kartal D, Dogan A, Ertas SK, Aydogdu EG, et al. (2016). "Androgenetic alopecia as an indicator of metabolic syndrome and cardiovascular risk". Blood Pressure. 25 (3): 141–148. doi:10.3109/08037051.2015.1111021. PMID26585114. S2CID12031777.
^Acibucu F, Kayatas M, Candan F (2010). "The association of insulin resistance and metabolic syndrome in early androgenetic alopecia". Singapore Medical Journal. 51 (12): 931–936. PMID21221497.
^Pengsalae N, Tanglertsampan C, Phichawong T, Lee S (2013). "Association of early-onset androgenetic alopecia and metabolic syndrome in Thai men: A case-control study". Journal of the Medical Association of Thailand = Chotmaihet Thangphaet. 96 (8): 947–951. PMID23991602.