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Trade names | Redux |
Other names | Dextrofenfluramine; d-Fenfluramine; (S)-Fenfluramine; S-Fenfluramine; (+)-Fenfluramine; S(+)-Fenfluramine; (S)-(+)-Fenfluramine |
MedlinePlus | a682088 |
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Pharmacokinetic data | |
Protein binding | 36% |
Metabolites | Dexnorfenfluramine |
Elimination half-life | 17–20 hours |
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Chemical and physical data | |
Formula | C12H16F3N |
Molar mass | 231.262 g·mol−1 |
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Dexfenfluramine, marketed as dexfenfluramine hydrochloride under the name Redux, is a serotonergic anorectic drug: it reduces appetite by increasing the amount of extracellular serotonin in the brain.[3] It is the d-enantiomer of fenfluramine and is structurally similar to amphetamine, but lacks any psychologically stimulating effects.
Dexfenfluramine was, for some years in the mid-1990s, approved by the United States Food and Drug Administration for the purposes of weight loss. However, following multiple concerns about the cardiovascular side-effects of the drug,[3] the FDA withdrew the approval in 1997.[4] After it was removed in the US, dexfenfluramine was also pulled out in other global markets. It was later superseded by sibutramine, which, although initially considered a safer alternative to both dexfenfluramine and fenfluramine,[5][6][7] was likewise removed from the US market in 2010.[8][9]
The drug was developed by Interneuron Pharmaceuticals, a company co-founded by Richard Wurtman, aimed at marketing discoveries by Massachusetts Institute of Technology scientists.[10] Interneuron licensed the patent to Wyeth-Ayerst Laboratories.[11] Although at the time of its release, some optimism prevailed that it might herald a new approach,[12] there remained some reservations amongst neurologists, twenty-two of whom petitioned the FDA to delay approval.[citation needed] Their concern was based on the work of George A. Ricaurte, whose techniques and conclusions were later questioned.[13]
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