Diabetic foot ulcer | |
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Neuropathic diabetic foot ulcer | |
Causes | diabetes |
Diabetic foot ulcer is a breakdown of the skin and sometimes deeper tissues of the foot that leads to sore formation. It is thought to occur due to abnormal pressure or mechanical stress chronically applied to the foot, usually with concomitant predisposing conditions such as peripheral sensory neuropathy, peripheral motor neuropathy, autonomic neuropathy or peripheral arterial disease.[1] It is a major complication of diabetes mellitus, and it is a type of diabetic foot disease. Secondary complications to the ulcer, such as infection of the skin or subcutaneous tissue, bone infection, gangrene or sepsis are possible, often leading to amputation.[1]
A key feature of wound healing is stepwise repair of lost extracellular matrix (ECM), the largest component of the dermal skin layer.[2] However, in some cases, physiological insult or disorder - in this case, diabetes mellitus - impedes the wound healing process. In diabetic wounds, the inflammatory phase of the healing process is prolonged, delaying the formation of mature granulation tissue and reducing the healing wound's tensile strength.[3]
Treatment of diabetic foot ulcers includes blood sugar control, removal of dead tissue from the wound, wound dressings, and removing pressure from the wound through techniques such as total contact casting. Surgery in some cases may improve outcomes.[4] Hyperbaric oxygen therapy may also help but is expensive.[4]
34% of people with diabetes develop a diabetic foot ulcer during their lifetime, and 84% of all diabetes-related lower-leg amputations are associated with or result from diabetic foot ulcers.[5]
Armstrong 2023
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was invoked but never defined (see the help page).