| Gerstmann syndrome | |
|---|---|
 | |
| The inferior parietal lobule, with damage in this area most often associated with Gerstmann syndrome | |
| Specialty | Neurology, neuropsychology  |
| Symptoms | Dysgraphia, dyscalculia, finger agnosia, left-right disorientation, constructional apraxia, aphasia |
| Causes | Idiopathic, stroke, dementia |
Gerstmann syndrome is a neuropsychological disorder that is characterized by a constellation of symptoms[1] that suggests the presence of a lesion usually near the junction of the temporal and parietal lobes at or near the angular gyrus. Gerstmann syndrome is typically associated with damage to the inferior parietal lobule of the dominant hemisphere. It is classically considered a left-hemisphere disorder, although right-hemisphere damage has also been associated with components of the syndrome.[2]
It is named after Jewish Austrian-born American neurologist Josef Gerstmann.[3]
- ^ João RB, Filgueiras RM, Mussi ML, de Barros JE (June 2017). "Transient Gerstmann syndrome as manifestation of stroke: Case report and brief literature review". Dementia & Neuropsychologia. 11 (2): 202–205. doi:10.1590/1980-57642016dn11-020013. PMC 5710689. PMID 29213512.
- ^ Heimburger RF, Demyer W, Reitan RM (February 1964). "Implications of Gerstmann's syndrome". Journal of Neurology, Neurosurgery, and Psychiatry. 27 (1): 52–57. doi:10.1136/jnnp.27.1.52. PMC 495679. PMID 14123925.
- ^ synd/2267 at Who Named It?