LY-2183240 is a drug which acts both as a potent inhibitor of the reuptake of the endocannabinoidanandamide and as an inhibitor of fatty acid amide hydrolase (FAAH), the primary enzyme responsible for degrading anandamide. This leads to markedly elevated anandamide levels in the brain, and LY-2183240 has been shown to produce both analgesic and anxiolytic effects in animal models.[1][2][3] While LY-2183240 is a potent inhibitor of FAAH, it has relatively poor selectivity and also inhibits several other enzyme side targets.[4] Consequently, it was never developed for clinical use, though it remains widely used in research, and has also been sold as a designer drug.[5]
^Dickason-Chesterfield AK, Kidd SR, Moore SA, Schaus JM, Liu B, Nomikos GG, Felder CC (2006). "Pharmacological characterization of endocannabinoid transport and fatty acid amide hydrolase inhibitors". Cellular and Molecular Neurobiology. 26 (4–6): 407–23. doi:10.1007/s10571-006-9072-6. PMID16736384. S2CID24361518.{{cite journal}}: CS1 maint: multiple names: authors list (link)
^Alexander JP, Cravatt BF (Aug 2006). "The putative endocannabinoid transport blocker LY2183240 is a potent inhibitor of FAAH and several other brain serine hydrolases". Journal of the American Chemical Society. 128 (30): 9699–704. doi:10.1021/ja062999h. PMID16866524.
^Uchiyama N, Matsuda S, Kawamura M, Shimokawa Y, Kikura-Hanajiri R, Aritake K, Urade Y, Goda Y (2014). "Characterization of four new designer drugs, 5-chloro-NNEI, NNEI indazole analog, α-PHPP and α-POP, with 11 newly distributed designer drugs in illegal products". Forensic Sci Int. 243: 1–13. doi:10.1016/j.forsciint.2014.03.013. PMID24769262.{{cite journal}}: CS1 maint: multiple names: authors list (link)