It was identified among the verified anti-inflammatory components in an extract of Sinomenii caulis[4] and has been proposed to have other potential physiological effects, such as sedative and anxiolytic,[2] reduction of erythrocyte hemolysis,[5] antifungal activity,[6] improvement of LPS-induced acute lung injury,[7] and protection against muscle atrophy.[8] Furthermore, magnoflorine has been identified to be an inhibitor of NF-κB activation and to be an agonist at the β2 -adrenergic receptor.[9]
(S)-Magnoflorine is metabolically derived from (S)-reticuline, a pivotal intermediate in the biosynthesis of numerous BIA structural subgroups, through two enzymatic steps: first, (S)-corytuberine synthase/CYP80G2 to (S)-corytuberine, and secondly, (S)-corytuberine-N-methyltransferase to (S)-magnoflorine.[10][11]
^El-Kawi, M. A.; Slatkin, D. J.; Schiff, P. L.; Dasgupta, S; Chattopadhyay, S. K.; Ray, A. B. (1984). "Additional alkaloids of Pachygone ovata". J Nat Prod. 47 (3): 459–64. doi:10.1021/np50033a010. PMID6481360.
^ abde la Peña, June Bryan I.; Lee, Hye Lim; Yoon, Seo Young; Kim, Gun Hee; Lee, Yong Soo; Cheong, Jae Hoon (Oct 2013). "The involvement of magnoflorine in the sedative and anxiolytic effects of Sinomeni Caulis et Rhizoma in mice". J Nat Med. 67 (4): 814–21. doi:10.1007/s11418-013-0754-3. PMID23456265. S2CID14170353.
^Lee, Heyjin; Tuong, Le Thi; Jeong, Ji Hye; Lee, Sang-Jin; Bae, Gyu-Un; Ryu, Jae-Ha (2017). "Isoquinoline alkaloids from Coptis japonica stimulate the myoblast differentiation via p38 MAP-kinase and Akt signaling pathway". Bioorg Med Chem Lett. 27 (6): 1401–1404. doi:10.1016/j.bmcl.2017.02.003. PMID28228365.
^Sun, Dan; Han, Yanqi; Wang, Weiya; Wang, Zengyong; Ma, Xiaoyao; Hou, Yuanyuan; Bai, Gang (2016). "Screening and identification of Caulis Sinomenii bioactive ingredients with dual-target NF-κB inhibition and β2-AR agonizing activities". Biomed Chromatogr. 30 (11): 1843–1853. doi:10.1002/bmc.3761. PMID27187693.