Mönckeberg's sclerosis | |
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Other names | Medial calcific sclerosis or Mönckeberg medial sclerosis |
Microphotography of arterial wall with calcified (violet colour) atherosclerotic plaque (haematoxylin & eosin stain) | |
Specialty | Cardiology |
Mönckeberg's arteriosclerosis, or Mönckeberg's sclerosis, is a non-inflammatory form of arteriosclerosis (artery hardening), which differs from atherosclerosis traditionally. Calcium deposits are found in the muscular middle layer of the walls of arteries (the tunica media)[1] with no obstruction of the lumen. [2] It is an example of dystrophic calcification. This condition occurs as an age-related degenerative process. However, it can occur in pseudoxanthoma elasticum and idiopathic arterial calcification of infancy as a pathological condition, as well. Its clinical significance and cause are not well understood and its relationship to atherosclerosis and other forms of vascular calcification are the subject of disagreement.[3] Mönckeberg's arteriosclerosis is named after Johann Georg Mönckeberg,[4][5] who first described it in 1903.
The severity of calcium deposits formed by Mönckeberg's arteriosclerosis can be categorized into stages based on the histological appearance. Understanding these stages can help to understand disease progression and how the disease is caused. Stage 1 involves the formation of calcium deposits both inside and outside the vascular smooth muscle cells which compose the tunica media. Calcification outside of the vascular smooth muscle cells are commonly associated with damage to elastic fibers in the extra-cellular matrix. These calcium deposits also develop on the internal elastic lamina. Stage 2 and Stage 3 involve the formation of calcified sheaths spanning an increased diameter through the tunica media. Stage 4 then involves the formation of bony tissue from these calcifications through the process of osseous metaplasia.[6][7]