Non-celiac gluten sensitivity | |
---|---|
Other names | Gluten sensitivity |
Specialty | Gastroenterology, internal medicine, neurology[1] |
Symptoms | Irritable bowel syndrome-like symptoms, fatigue, headache, fibromyalgia, atopic disorders, neurological diseases, psychiatric problems[2][3][4][5][6][7] |
Usual onset | Any age[8] |
Duration | lifelong[9] |
Causes | Reaction to gluten, other proteins and FODMAPS from gluten-containing cereals[3][10] |
Diagnostic method | Exclusion of celiac disease and wheat allergy, improvement with gluten withdrawal and worsening after gluten consumption[6][11][12] |
Treatment | Gluten-free diet |
Frequency | 0.5–13%[13] |
Non-celiac gluten sensitivity (NCGS) or gluten sensitivity[14] is a controversial disorder which can cause both gastrointestinal and other problems.
NCGS is included in the spectrum of gluten-related disorders.[3][4] The definition and diagnostic criteria of non-celiac gluten sensitivity were debated and established by three consensus conferences.[4][14][15][16][17] However, as of 2019[update], there remained much debate in the scientific community as to whether NCGS was a distinct clinical disorder.[18]
The pathogenesis of NCGS is not well understood, but the activation of the innate immune system, the direct cytotoxic effects of gluten and probably other wheat components, are implicated.[3][19][20] There is evidence that not only gliadin (the main cytotoxic antigen of gluten), but also other proteins named ATIs which are present in gluten-containing cereals (wheat, rye, barley, and their derivatives) may have a role in the development of symptoms. ATIs are potent activators of the innate immune system.[3][21] FODMAPs, especially fructans, are present in small amounts in gluten-containing grains and have been identified as a possible cause of some gastrointestinal symptoms in NCGS patients.[3][10][22][21] As of 2019[update], reviews have concluded that although FODMAPs may play a role in NCGS, they explain only certain gastrointestinal symptoms, such as bloating, but not the extra-digestive symptoms that people with NCGS may develop, such as neurological disorders, fibromyalgia, psychological disturbances, and dermatitis.[21][9][3]
For these reasons, NCGS is a controversial clinical condition[23] and some authors still question it.[24][25] It has been suggested that "non-celiac wheat sensitivity" is a more appropriate term, without forgetting that other gluten-containing cereals are implicated in the development of symptoms.[11][24]
NCGS is the most common syndrome of gluten-related disorders[4][26] with prevalence rates between 0.5–13% in the general population.[13] As no biomarker for diagnosing this condition is available, its diagnosis is made by exclusion of other gluten-related disorders such as celiac disease and wheat allergy.[23][27] Many people have not been diagnosed following strict criteria, and there is a "fad component" to the recent rise in popularity of the gluten-free diet, leading to debate surrounding the evidence for this condition and its relationship to celiac disease and irritable bowel syndrome.[3][5] People with NCGS are often unrecognized by specialists and lack adequate medical care and treatment.[28] They often have a long history of health complaints and unsuccessful consultations with physicians, and thus many resort to a gluten-free diet and a self-diagnosis of gluten sensitivity.[29]
Cereals such as wheat and rye, when consumed in normal quantities, are only minor sources of FODMAPs in the daily diet. (...) Table 1. Sources of FODMAPs (...) Oligosaccharides (fructans and/or galactans). Cereals: wheat and rye when eaten in large amounts (eg, bread, pasta, couscous, crackers, biscuits)
CatassiBai2013
was invoked but never defined (see the help page).LebwoholLudvigsson
was invoked but never defined (see the help page).VoltaCaio2015
was invoked but never defined (see the help page).AzizHadjivassiliou2015
was invoked but never defined (see the help page).VoltaDeGiorgio2019
was invoked but never defined (see the help page).VoltaCaioQuestions
was invoked but never defined (see the help page).GreenLebwohl
was invoked but never defined (see the help page).Since 2010, the definition of NCGS has been discussed at 3 consensus conferences, which led to 3 publications. Given the uncertainties about this clinical entity and the lack of diagnostic biomarkers, all 3 reports concluded that NCGS should be defined by the following exclusionary criteria: a clinical entity induced by the ingestion of gluten leading to intestinal and/or extraintestinal symptoms that resolve once the gluten-containing foodstuff is eliminated from the diet, and when celiac disease and wheat allergy have been ruled out.
According to the diagnostic criteria established by two Consensus Conferences (London 2011 and Munich 2012), the current view to NCGS diagnosis is based on symptom / manifestation evaluation along with the exclusion of CD and WA [5,7].
However, there is a great deal of skepticism within the scientific community questioning the existence of NCGS as a distinct clinical disorder. There are no strict diagnostic criteria and a placebo-controlled rechallenge trial has been recommended for diagnosis
ElliRoncoroni2015
was invoked but never defined (see the help page).LeonardSapone2017
was invoked but never defined (see the help page).Verbeke2018
was invoked but never defined (see the help page).OntiverosHardy
was invoked but never defined (see the help page).NCGS is a clinical condition in which intestinal and extraintestinal symptoms are triggered by gluten ingestion, in the absence of coeliac disease and wheat allergy. The symptoms usually occur soon after gluten ingestion, improve or disappear within hours or a few days after gluten withdrawal, and relapse following its reintroduction. ... Unlike coeliac disease and wheat allergy, NCGS is an unclear and controversial entity.
One of the most controversial and highly debated discussions concerns the role of gluten in causing NCGS. Recent reports have indicated that gluten might not be the cause of NCGS, and some investigators still question whether NCGS as a real clinical entity. (...) Cereals such as wheat and rye, when consumed in normal quantities, are only minor sources of FODMAPs in the daily diet (Table 1). Therefore, gluten-containing grains are not likely to induce IBS exclusively via FODMAPs. In contrast, there is growing evidence that other proteins that are unique to gluten-containing cereals can elicit an innate immune response that leads to NCGS, raising a nomenclature issue. For this reason, wheat sensitivity, rather than gluten sensitivity, seems to be a more appropriate term, keeping in mind that other gluten-containing grains such as barley and rye also can trigger the symptoms.
The new syndrome has been named non-celiac gluten sensitivity (NCGS) or gluten sensitivity (GS).
mansueto-etal-2014
was invoked but never defined (see the help page).