Stephen L. Hauser

This article has multiple issues. Please help improve it or discuss these issues on the talk page. (Learn how and when to remove these messages) A major contributor to this article appears to have a close connection with its subject. It may require cleanup to comply with Wikipedia's content policies, particularly neutral point of view. Please discuss further on the talk page. (October 2016) (Learn how and when to remove this message) ‹ The template below (Promotional) is being considered for merging. See templates for discussion to help reach a consensus. › This article contains promotional content. Please help improve it by removing promotional language and inappropriate external links, and by adding encyclopedic text written from a neutral point of view. (October 2016) (Learn how and when to remove this message) The topic of this article may not meet Wikipedia's notability guideline for academics. Please help to demonstrate the notability of the topic by citing reliable secondary sources that are independent of the topic and provide significant coverage of it beyond a mere trivial mention. If notability cannot be shown, the article is likely to be merged, redirected, or deleted. Find sources: "Stephen L. Hauser" – news · newspapers · books · scholar · JSTOR (September 2022) (Learn how and when to remove this message) (Learn how and when to remove this message)

Stephen L. Hauser
Born	(1949-12-14) December 14, 1949 (age 74) New York, New York, U.S.
Nationality	American
Alma mater	Harvard Medical School
Known for	multiple sclerosis research bioethics
Awards	John Dystel Prize for Multiple Sclerosis Research (2008) Charcot Award (2013)
Scientific career
Fields	Neurology, neuroimmunology
Institutions	University of California, San Francisco

Stephen L. Hauser is a professor of the Department of Neurology at the University of California, San Francisco (UCSF) specializing in immune mechanisms and multiple sclerosis (MS). He has contributed to the establishment of consortia that have identified more than 50 gene variants that contribute to MS risk.^[1]